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The Last Piece of the Puzzle

The Logic Behind the Viral Connection

  1. If one is going to clearly understand something like autism, one has to be logical. It has to make sense in the world as we know it. Mysterious, alien, puzzling and so on are not words that fit a biological reality even though they commonly used to describe autism..
  2. The cause of autism is not simple and will never be traced to one single factor. If it were so, we would have found it many years ago.
  3. The cause of autism HAS to fit every single case if this is one condition. We can't pick and choose the cases that are going to support our position. There was a time that I believed that we may be throwing a lot of different conditions into one basket and giving them one name. I do not believe that any more.
  4. We do have documented cases of autism in children who have never been vaccinated, so the actual vaccinations and/or the mercury or aluminium in vaccinations cannot be the "cause" of autism.
  5. The symptoms of mercury poisoning and the symptoms of autism are similar, but not the same, so mercury poisoning, whether through environmental exposure or the vaccination process cannot be the "cause" of autism. This does not mean that people on the autism spectrum may not also have a build-up of mercury in their bodies from a number of different sources including vaccinations.
  6. The symptoms of aluminium poisoning and the symptoms of autism are similar, but not the same, so aluminium poisoning, whether through environmental exposure or the vaccination process cannot be the "cause" of autism.
  7. The incidence of autism has increased with the advent of the vaccination process and continues to increase as we add more and more vaccinations to the bodies of our little children which indicates that they may be involved.
  8. The incidence of autism continues to increase in spite of the fact that thimerosal (the source of mercury in vaccinations) has been eliminated and/or reduced in the vaccines.
  9. The incidence of autism has not increased in certain populations such as the Amish, who do not vaccinate their children.
  10. There is a lot research which indicates that autism runs in families. In other words, there is a genetic component.
  11. If you read the biographies of the adults and parents of the older adults (pre-vaccination age) on the spectrum you will find that there is usually a description of an exposure to a virus during the pregnancy. Discussions with the older adults also reveal the same thing. There is not one specific virus that appears in every case.
  12. If you read the biographies of the younger people on the spectrum and their parents you will find that there is either a description of an exposure to a virus during pregnancy and/or a heightened reaction and regression after a vaccination. You will find the same if you listen to the parents and to individuals on the spectrum.
  13. Although the majority of autism cases at the present moment appear to be vaccine related, we still have those in which mothers recognize the symptoms at birth which negates vaccinations as the "cause".
  14. Research has indicated that there appears to be a connection between autism and exposure to specific viruses over the years. Much of this work has been ignored because it does not explain every single case.
  15. Research has indicated that there is a difference in antibodies and immune co-factors carried by individuals with autism in regards to specific viruses when compared to typical subjects. Again, much of this work has been ignored because it does not explain every case.
  16. My work with the SCIO has indicated specific viruses for each individual as the source of the autism for the individual. The type and number of viruses that come up are unique to each individual.
  17. Family members are often able to pinpoint the connection to specific viruses for the child in their family history. For example: "His maternal grandmother had polio."
  18. When we clear the viruses from the body, the autistic symptoms decrease: at times instantly and in other cases more gradually as we deal with the other trauma that has happened to the body over time.

A link to a video presentation which describes research into the viral connection with autism in detail:  http://www.chem.cmu.edu/wakefield/

A Short List of References Available

Chess, S. (1971). Autism in children with congenital rubella. J. Autism Child. Schizophr. 1, 33-47.

Chess, S. (1977). Follow-up report on autism in congenital rubella. J. Autism Child Schizophr. 7, 69-81.

Casanova, MF (2008): The minicolumnopathy of autism: a link between migraine and gastrointestinal symptoms. Med Hypothesis. 70(1): 73-80.

Casanova, M. F., Buxhoeveden, D. P., Switala, A. E., and Roy, E. (2002). Minicolumnar pathology in autism. Neurology 58, 428-432.

Cook, E.H., and Jr. (2001). Genetics of autism. Child Adolesc. Psychiatr. Clin. N. Am. 10, 333-350.

Fatemi SH, Folsom TD, Reutiman TJ, Abu-Odeh D, Mori S, Huang H, Oishi K. (2009). Abnormal expression of myelination genes and alternations in white matter fractional anisotropy following prenatal viral influenza infection at E16 in mice. Schizophrenia Research. Jul:112(1-3), 46-53.

Fatemi SH, Reutiman TJ, Folsom TD, Huang H, Oishi K, Mori S, Smee DF, Pearce DA, Winter C, Sohr R, Juckel G. (2008).  Maternal infection leads to abnormal gene regulation and brain atrophy in mouse offspring: implications for genesis of neurodevelopmental disorders. Schizophrenia Research. Feb: 99(1-3), 56-70.

Hallmayer, J., Glasson, E. J., Bower, C., Petterson, B., Croen, L., Grether, J., and Risch, N. (2002). On the twin risk in autism. Am. J. Hum. Genet 71: 941-946.

Lancaster K, Dietz DM, Moran TH, Pletnikov MV. (2007). Abnormal social behaviours in young and adult rats neonatally infected with Borna disease virus. Behaviour and Brain Research: Jan 10: 176(1), 141-148.

Lui XQ, Paterson AD, Szatmari P, Autism Genome Project Consortium. (2008). Genome-wide linkage analyses of quantitative and categorical autism subphenotypes. Biological Psychiatry. Oct. 1:64(7), 561-570.

Merikangas AK, Corvin AP, Gallagher L (2009). Copy-number variants in neurodevelopmental disorders: promises and challenges. Trends in Genetics: Nov 10.

Nicolson GL, Gan R, Nicolson NL, Haler J. (2007).  "Evidence for mycoplasma ssp., chlamydia pneunomiae, and human herpes virus-6 coinfections in the blood of patients with autistic spectrum disorders. Journal of Neuroscience Research. April: 85(5), 1143-1148.

Singh, VK (2009). "Phenotypic expression of autimmune autistic disorder (AAD): a major subset of autism". Ann Clin Psychiatry. Jul-Sep: 21 (3):148-161.

Singh VK, Lin SX, Newell E, Nelson C (2002). "Abnormal measles-mumps-rubella antibodies and CNS autoimmunity in children with autism". J Biomed Sci 9 (4): 359-64

Singh, VK, Lin, SX and Yang, VC. (1998) "Serological association of measles virus and human herpes virus-6 with Brain autoantibodies in autism". Clinical Immunology and Immunopathology. Oct: 89(1), 105-108.

Shi L, Smith SE, Malkova N, Tse D, Su Y, Patterson PH. (2009). Activation of the maternal immune system alters cerebellar development in the offspring. Brain Behaviour Immunology. Jan:23(1), 116-123.

Wakefield AJ.(2002). Enterocolitis, autism and measles virus. Mol. Psychiatry 7:Supplement, 2:S444-S446, Review.

Weiss LA. (2009) Autism genetics: emerging data from genome-wide copy-number and single nucleotide polymorphism scans. Expert Rev Mol Diagn. Nov: 9(8), 795-803.

Winter C, Reutiman TJ, Folsom TD, Sohr R, Wolf RJ, Juckel G, Fatemi SH (2008). "Dopamine and serotonin levels following prenatal viral infection in mouse - implications for psychiatric disorders such as schizophrenia and autism". Eur Neuropsychopharmacol. Oct: 18(10), 712-6.

 

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Last modified: 11/12/09